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An Exciting Partnership!
Cellular Microbiology is part of a partnership between Wiley and Hindawi and is now fully open access. Cellular Microbiology will remain a Wiley title but will be published and hosted by Hindawi and will benefit from Hindawi’s experience and expertise in publishing open access titles. Cellular Microbiology will continue to undergo a rigorous peer review process ensuring that quality remains high. Manuscripts published after January 1, 2022 will be published as open access articles, making them immediately free to read, download and share. Authors or their funder will be required to pay an Article Publication Charge upon acceptance. For further information, click here.
Virtual Issue - Reactive Oxygen species in the interface of host-pathogen interactions
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Virtual Issue - Pathogen evasion strategies
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Virtual Issue - Intracellular pathogens, membrane damage and cytosolic access
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Thank you to our TOP reviewers 2020!
Reviewers play an essential role in shaping the content of the journal, guarantee the high standard of our publications and so strongly influence the impact of the journal.
Join us in thanking our reviewers for their valuable contributions.
Click here for a list of our TOP reviewers.
Reasons to publish in Cellular Microbiology
- here is no need to reformat your manuscript to submit it to the journal
- Expedited peer review - making a decision quickly where possible on your previous peer reports when provided
- Promotion of your paper through the Cellular Microbiology twitter account @CellMicrobiol
- Free to publish - no colour or page charges for your article
- Friendly and helpful Editorial Board offers guidance through the peer review process
Our publication metrics
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Do you wish to publish your research in Cellular Microbiology?
We are awaiting your submission at https://mc.manuscriptcentral.com/cell-micro
Please make sure to follow our author guidelines during the preparation of your manuscript. Please note that these have been updated recently. Cellular Microbiology requests now Graphical Abstract and Take Away for most article types.
The Graphical Abstracts will be displayed in the table of contents. These images are meant to attract reader interest and may encapsulate a key result or conclusion of the paper. Appropriate images might include molecular or cellular structures, appealing graphics, or models or summary figures. A figure (or panel of a figure) from the main text may be used, if appropriate, or an image may be prepared specifically for this purpose.
The Take Away summarizes the main points of your article. It needs to be included in the article itself and should outline 3-5 major points/key findings to attract the reader’s attention and to enhance the article’s placing in search engines. Please limit each point to maximal 80 characters (including spaces). The Take Away should attract readers to your article as well as let them remember your main results.
Upgraded Data Sharing Policy
Cellular Microbiology expects data sharing. All accepted manuscripts are required to publish a data availability statement to confirm the presence or absence of shared data. Review Wiley’s Data Sharing policy where you will be able to see and select the data availability statement that is right for your submission. Please find here standard templates for the data availability statement to be used in your manuscript. While it is optional for initial submissions, a data availability statement is required when submitting your revised manuscript.
While submitting your manuscript, please make sure to include the data availability statement in the submission system as well as in your main manuscript file. During the submission process, you will find the relevant field to enter the data availability statement in “Step 6: Details & Comments”.
Vam6/Vps39/TRAP1-domain proteins influence vacuolar morphology, iron acquisition and virulence in Cryptococcus neoformans
-  20 November 2021
The pathogenic fungus Cryptococcus neoformans must overcome iron limitation to cause disease in mammalian hosts. Two Vam6/Vps39/TRAP1 domain-containing proteins, Vps3 and Vam6, are required for robust growth on haem as well as proper regulation of iron homeostasis. Two independent deletion mutants for each gene, or double mutants lacking both genes, are unable to cause disease in a mouse inhalation model of cryptococcosis.
Hepatitis B virus envelope proteins can serve as therapeutic targets embedded in the host cell plasma membrane
-  3 November 2021
Surface immunoprecipitation of HBS confirmed that membrane-associated HBs remains correctly folded in HBV-replicating cells in cell culture. MoMab coated onto superparamagnetic iron oxide nanoparticles allowed to detect membrane-associated HBs after HBV infection by electron microscopy in distinct stretched of the hepatocyte plasma membrane. Last but not least, we demonstrate that HBs located on the cell surface allow therapeutic targeting of HBV-positive cells by T-cells either engrafted with a chimeric antigen receptor or redirected by bispecific, T-cell engager antibodies.
Chlamydia and HPV induce centrosome amplification in the host cell through additive mechanisms
-  30 October 2021
HPV and Chlamydia have additive effects on the prevalence of centrosome amplification when present in the same host cell. While HPV primarily causes centriole overduplication through expression of the oncoprotein E7, Chlamydia causes cytokinesis defects, which then lead to centrosome amplification and multinucleation in the host cell.
Entry of the Varicellovirus Canid herpesvirus 1 into Madin–Darby canine kidney epithelial cells is pH-independent and occurs via a macropinocytosis-like mechanism but without increase in fluid uptake
-  25 October 2021
The entry pathway of Canid herpesvirus 1 into MDCK cells was shown to be via a macropinocytosis-like mechanism. CHV-1 induces membrane ruffles and internalises within large uncoated endocytic vacuoles. Pharmacological inhibitors targeting the macropinocytic machinery blocked virus entry, while in contrast, inhibiting endosomal acidification had no effect on the ability of the virus to infect MDCK cells.
Dengue virus replication enhances labile zinc pools by modulation of ZIP8
-  7 October 2021
The following is a list of the most cited articles based on citations published in the last three years, according to CrossRef.
Defining dysbiosis and its influence on host immunity and disease
-  5 May 2014
Lounging in a lysosome: the intracellular lifestyle of Coxiella burnetii
-  25 January 2007
Caspase‐1‐dependent pore formation during pyroptosis leads to osmotic lysis of infected host macrophages
-  4 July 2006
The role of the lung microbiota and the gut–lung axis in respiratory infectious diseases
-  17 October 2018
Neutrophil extracellular traps capture and kill Candida albicans yeast and hyphal forms
-  28 November 2005
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